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 Previous Article

Antimicrobial Agents and Chemotherapy, May 1999, p. 1314-1316, Vol. 43, No. 5
0066-4804/99/$04.00+0

Sensitivity of Spermidine-Deficient Saccharomyces cerevisiae to Paromomycin

David Balasundaram, Celia White Tabor, and Herbert Tabor*

National Institutes of Health, Bethesda, Maryland 20892-0830

Received 14 August 1998/Returned for modification 27 January 1999/Accepted 5 March 1999

Spermidine-deficient Saccharomyces cerevisiae cells are much more sensitive to paromomycin than nondeficient cells, resulting in cessation of growth and cell death.

* Corresponding author. Mailing address: NIH-NIDDK, Building 8, Room 225, Bethesda, MD 20892-0830. Phone: (301) 496-2562. Fax: (301) 402-0240. E-mail: tabor{at}helix.nih.gov.

Antimicrobial Agents and Chemotherapy, May 1999, p. 1314-1316, Vol. 43, No. 5
0066-4804/99/$04.00+0


This article has been cited by other articles:

  • Chattopadhyay, M. K., Park, M. H., Tabor, H. (2008). Hypusine modification for growth is the major function of spermidine in Saccharomyces cerevisiae polyamine auxotrophs grown in limiting spermidine. Proc. Natl. Acad. Sci. USA 105: 6554-6559 [Abstract] [Full Text]  
  • Chattopadhyay, M. K., Tabor, C. W., Tabor, H. (2002). Absolute requirement of spermidine for growth and cell cycle progression of fission yeast (Schizosaccharomycespombe). Proc. Natl. Acad. Sci. USA 99: 10330-10334 [Abstract] [Full Text]  


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