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Antimicrobial Agents and Chemotherapy, December 2004, p. 4882-4885, Vol. 48, No. 12
0066-4804/04/$08.00+0     DOI: 10.1128/AAC.48.12.4882-4885.2004
Copyright © 2004, American Society for Microbiology. All Rights Reserved.

VanE-Type Vancomycin-Resistant Enterococcus faecalis Clinical Isolates from Australia

Lorena Abadía-Patiño,1,{dagger} Keryn Christiansen,2 Jan Bell,3 Patrice Courvalin,1 and Bruno Périchon1*

Unité des Agents Antibactériens, Institut Pasteur, Paris, France,1 Department of Microbiology and Infectious Diseases, Royal Perth Hospital, Perth, Western Australia,2 Women's and Children's Hospital, Adelaide, South Australia, Australia3

Received 22 June 2004/ Returned for modification 16 July 2004/ Accepted 9 August 2004

Three distinct Enterococcus faecalis VanE-type isolates—BM4574, BM4575, and BM4576-obtained in Australia were studied. Expression of the resistance genes was constitutive in BM4575, probably due to a 2-bp deletion into the vanSE gene, and inducible in BM4574 and BM4576. Transcription analysis of the vanE operons suggested that the five genes were cotranscribed from an initiation site located 25 bp upstream from the ATG start codon of vanE.

* Corresponding author. Mailing address: Unité des Agents Antibactériens, Institut Pasteur, 25, Rue du Dr. Roux, 75724 Paris Cedex 15, France. Phone: (33) (1) 45-68-83-18. Fax: (33) (1) 45-68-83-19. E-mail: brunoper{at}pasteur.fr.

{dagger} Present address: IIBCA Universidad de Oriente, Biomedica, Cerro del Medio, 6101 Cumaná, Edo. Sucre, Venezuela.

Antimicrobial Agents and Chemotherapy, December 2004, p. 4882-4885, Vol. 48, No. 12
0066-4804/04/$08.00+0     DOI: 10.1128/AAC.48.12.4882-4885.2004
Copyright © 2004, American Society for Microbiology. All Rights Reserved.



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