RT Journal Article
SR Electronic
T1 SOS Regulation of <em>qnrB</em> Expression
JF Antimicrobial Agents and Chemotherapy
JO Antimicrob. Agents Chemother.
FD American Society for Microbiology
SP 821
OP 823
DO 10.1128/AAC.00132-08
VO 53
IS 2
A1 Wang, Minghua
A1 Jacoby, George A.
A1 Mills, Debra M.
A1 Hooper, David C.
YR 2009
UL http://aac.asm.org/content/53/2/821.abstract
AB In the sequence upstream from qnrB (but not qnrA or qnrS) is a LexA binding site. qnrB was shown to be under SOS control by demonstrating that quinolone susceptibility decreased with increasing temperature in a strain with a recA441(Ts) allele, whereas qnrB expression increased in response to ciprofloxacin or mitomycin C in strains with an intact lexA gene.