TABLE 1.

Strains used in this study and Southern hybridization data with various probes

StrainSerovarResistance phenotypeaGenomic island nameSize(s) (kb) of XbaI fragment(s) hybridizingb with:Reference
aadA2floRtet(G)pse-1qac/sul1tnpA/S044p1-9
96-5227DT104ACSSuTSGI111.711.711.711.7, 4.311.7, 4.34.39.0, 4.1 29
959SA97AgonaACSSuTSGI111.711.711.711.7, 4.311.7, 4.34.39.0, 4.1 10
1169SA97AgonaACSSuTTmSGI1-A11.711.711.711.7, 8.811.7, 8.88.89.0, 4.1 4
S/960725DT104ASuSGI1-B2.5, 4.34.34.39.0, 4.1 4
0047SA97AgonaSSuSGI1-C6.66.66.69.0, 4.1This study
S/954435DT104SSuSGI1-C6.66.66.69.0, 4.1 4
953SA98AgonaASSuTmcSGI1-DNDNDNDNDNDNDNDThis study
S/960081DT104ASSuTSGI1-E9.19.1, 7.99.19.1, 7.99.19.1, 7.99.0, 4.1 4
  • a A, ampicillin; C, chloramphenicol (and flofenicol); S, spectinomycin and streptomycin; Su, sulfonamides; T, tetracycline; Tm, trimethoprim.

  • b —, no hybridization signal observed; ND, not done.

  • c Ampicillin resistance is due to the presence of a TEM β-lactamase.